LEI 11418 PDF

LEI 11418 PDF

These food safe bags are perfect for holiday baked goods or handcrafted gifts. Each pack comes with 12 bags that measure 4 1/4″ x 9 1/2″ x 2 1/2″ and Type: Research Support, Non-U.S. Gov’t, Journal Article DOI: /s z The Digital Object Identifier (DOI) System enables identification of. Type: Journal Article DOI: /s The Digital Object Identifier (DOI) System enables identification of digital entities.

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Our data suggest that Leii has the potential to prevent insulin resistance and eli a promising anti-diabetic drug. Or filter your current search. We investigated the effects of GBE on glucose consumption, glucokinase activity, and mRNA levels of key genes in glucose metabolism and the insulin signaling pathway.

Journal of Natural Medicines [03 Sep65 1: Abstract Ginkgo biloba, an herbal medication, is capable of lowering glucose, fat, and lipid peroxide in diabetic patients. Gene Ontology GO Terms.

Find all citations in this journal default. In the current study, we tested the hypothesis that Ginkgo biloba extract GBE prevented hyperinsulinism-induced glucose intolerance in hepatocytes.

Find all citations in this journal default. Either your web browser doesn’t support Javascript or it is currently turned off. Body weight and BMD were both significantly decreased in osteoporotic rats, although BMD was partially restored by intraperitoneal morin administration. CitePeer Related Articles http: How does Europe PMC derive its citations network?

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Legal Entity Identifier – LEI Database Search

Furthermore, after analyzing gene expression, we suggest that GBE chiefly exerts its effects by stimulating IRS-2 transcription. Or filter your current search. Journal of Natural Medicines lek Jul72 4: To better show its efficacy, we included a control group that was treated with rosiglitazone, a type of thiazolidinedione TZD. It is said that GBE treatment could avoid drug-induced obesity.

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Here, we uncovered the therapeutic effect of morin against osteoporosis and demonstrated its suppressive action on the MAPK pathway in this disease. The data indicated that GBE repressed glucose uptake under normal conditions, while it dramatically improved glucose tolerance under insulin-resistant conditions.

Pathological examination was performed by hematoxylin and eosin staining. In the latter case, please turn on Javascript support in your web browser and reload this page. Morin treatment kei increased the number of trabecular bones in DEX-induced rats.

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Gene Ontology GO Terms.

It should be noted that, unlike rosiglitazone, GBE did not stimulate excessive glucose uptake as it improved glucose tolerance. The relative expression of osteogenic and bone resorption markers was determined by real-time polymerase chain reaction and Western blotting, respectively.

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Body weight was regularly monitored. Europe PMC requires Javascript to function effectively. Abstract Our aim is to investigate the potential therapeutic value of morin against osteoporosis and elucidate the mechanism of action. How does Europe PMC derive its citations network?

Body mineral density BMD was determined at proximal femurs using dual energy X-ray absorptiometry. CitePeer Related Articles http: Europe PMC requires Javascript to function effectively. Either your web browser doesn’t support Javascript or it is currently turned off. Mechanistically, morin reversed the decrease of osteogenic markers and increase of bone resorption markers, which might eventually 114118 mediated by modulation of MAPK signaling cascades.